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Beyond intracellular sensing via NODs, PGN-derived fragments function as potent signals that trigger the assembly of the NOD-like receptor protein 3 (NLRP3) inflammasome, leading to the maturation and secretion of IL-1

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Abstract Glucose-6-phosphate dehydrogenase (G6PD) deficiency, which involves the mutation of the G6PD gene on the X chromosome, is the most common enzyme defect in humans

However, Tang et al

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