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Interestingly, RSL3-induced ferroptosis could be inhibited by higher expression levels of mitochondrial ferritin under hypoxic conditions [60]

KIM-1, a biomarker of renal tubular damage, was examined by IHC staining

GSH-dependent activation releases Cu + to engage lipoylated DLAT and simultaneously unmasks glucose oxidase activity, inducing nutrient deprivation and triggering cuproptosis 216

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Moreover, as to our knowledge these data are the first to show a proliferative response in the Drosophila adult fat body in response to injury, these data provide evidence for a new modality to study liver and tissue regeneration in Drosophila